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Cardiac sodium channel NaV1.5 is the predominant form of sodium channels in cardiomyocytes, which exists as a macromolecular complex and interacts with multiple protein partners. Fyn kinase is one of the interacting proteins which colocalize, phosphorylate and modulate the NaV1.5 channel. To elaborate this interaction we created expression vectors for the N‐terminal, intracellular loop, and C‐terminal...