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The cellular cytidine deaminase APOBEC3G (A3G) was first described as an anti‐HIV‐1 restriction factor, acting by directly deaminating reverse transcripts of the viral genome. HIV‐1 Vif neutralizes the activity of A3G, primarily by mediating degradation of A3G to establish effective infection in host target cells. Lymphoma cells, which express high amounts of A3G, can restrict Vif‐deficient HIV‐1...
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