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MYCT1 is an important gene known to regulate cell viability and apoptosis of laryngeal cancer cells. However, the underlying molecular mechanism remains unclear. Here, we show that MAX enhances the expression of miR‐181a by directly binding to its promoter, whereas miR‐181a targets NPM1 and suppresses its expression in laryngeal cancer cells. MYCT1 and miR‐181a decrease cell viability and colony formation...
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