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Pathological features of Alzheimer’s disease (AD) include accumulation and deposition of β-amyloid (Aβ) in the brain, activation of astrocytes and microglia, and disruption of cholinergic neurotransmission. In our experiments on rats, a model of AD was created by intracerebroventricular (i.c.v.) injections of Aβ25–35 amyloid. In another animal group, this was combined with intramuscular (i.m.) injections...