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A unique defense mechanisms by which Mycobacterium tuberculosis protects itself from nitrosative stress is based on the O2‐dependent NO‐dioxygenase (NOD) activity of truncated hemoglobin 2/2HbN (Mt2/2HbN). The NOD activity largely depends on the efficiency of ligand migration to the heme cavity through a two‐tunnel (long and short) system; recently, it was also correlated with the presence at the...
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