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Ca2+‐dependent adhesion molecules, cadherins, localised at synaptic sites are critically involved in long‐term potentiation (LTP). N‐cadherin is thought to promote LTP whereas cadherin‐11 seems to counteract LTP. Since high synaptic activity is accompanied by local transient changes of the pH in the synaptic cleft, we studied whether the binding activity of cadherins is dependent on the pH and whether...