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Ras genes are frequently mutated in many cancer types; however, there are currently no conclusively effective anticancer drugs against Ras‐induced cancer. Therefore, the downstream effectors of Ras signaling need to be identified for the development of promising novel therapeutic approaches. We previously reported that oncogenic Ras induced the expression of NF‐HEV/IL‐33, a member of the interleukin‐1...
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