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Our previous work on the redox activation of DUSP4 by NAC in endothelial cells demonstrated that DUSP4 is an antioxidant protein, critical in the modulation of MAPK signal pathways and eNOS expression by protecting against oxidant-induced stress. More recently, we demonstrated that DUSP4-/- mouse hearts suffer greater infarct than wild-type (WT) and have poor post-ischemic functional recovery when...