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Les glomérulonéphrites inflammatoires graves dites « extracapillaires » (GNEC) sont étudiées grâce au modèle murin de glomérulonéphrite par anticorps anti-membrane basale glomérulaire (MBG). La pathogénie des lésions est classiquement attribuée aux effecteurs de l’immunité adaptative et en particulier aux lymphocytes T.Nous étudions le rôle des récepteurs de l’IL-15 exprimés par l’épithélium rénal...
Implantation sites from three strains of immunodeficient mice [tgϵ26, IL-2Rβ null×p56 lck null and IL-2Rγ null, now known as common cytokine chain γ (γ c ) null], which lack uterine natural killer (uNK) cells, are histologically abnormal. The related anomalies (found from day 10 of gestation) include the absence of aggregation of lymphocytes in the mesometrial triangle, acellularity...
Mice genetically deficient in the natural killer (NK) cell lineage lack uterine (uNK cells) and demonstrate morphometrically-quantifiable histopathology within their implantation sites. Two particular mouse strains, tgϵ,26 and RAG-2 null×γc null, have been used successfully as transplant recipients to address questions relating to the biology of uNK cells. uNK cells did not differentiate within decidualized...
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