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Myofibroblast apoptosis is essential for normal resolution of wound repair, including cardiac infarction repair. Impaired cardiac myofibroblast (CMF) apoptosis is associated with excessive extracellular matrix (ECM) deposition, which could be responsible for pathological cardiac fibrosis. Conventionally, angiotensin II (Ang II), a soluble peptide, is implicated in fibrogenesis because it induces cardiac...
The inflammatory receptor Toll-like receptors (TLRs) activation could induce endothelial inflammatory responses, which plays an important role in the development of many diseases including atherosclerosis. We already found that TLR2 activation of Peptidoglycan (PGN) stimulation could increase intercellular adhesion molecule-1 (ICAM-1) expression in HCAECs. Since anti-inflammatory cytokine interleukin...
IL-1 family member IL-37 functions to suppress innate inflammation in models of colitis, LPS-induced shock and ischemic myocardial injury. In the present study, we examined a role for IL-37 in the adaptive immune response, namely the development of skin contact hypersensitivity (CHS) to specific antigen. We generated a strain of transgenic mice expressing human IL-37 (IL-37Tg). Not expressed in the...
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