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J. Neurochem. (2012) 121, 629–638.
AbstractA corticostriatal‐dependent deficit in the release of ascorbate (AA), an antioxidant vitamin and neuromodulator, occurs concurrently in striatum with dysfunctional GLT1‐dependent uptake of glutamate in the R6/2 mouse model of Huntington’s disease (HD), an autosomal dominant condition characterized by overt corticostriatal dysfunction. To determine if deficient...
A behavior-related deficit in the release of ascorbate (AA), an antioxidant vitamin, occurs in the striatum of R6/2 mice expressing the human mutation for Huntington's disease (HD), a dominantly inherited condition characterized by striatal dysfunction. To determine the role of corticostriatal fibers in AA release, we combined slow-scan voltammetry with electrical stimulation of cortical afferents...
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