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This study evaluates the cellular responses to spinal cord injury in normal mice of the C57BL/6J strain and in a substrain of mice carrying the WLDs mutation which leads to a substantial delay in the onset of Wallerian degeneration in amputated axons. The spinal cord of normal C57BL6J and C57BL6J/WLD s mice was crushed at T8 using an extradural approach and animals were allowed to survive...