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L’hématurie et la protéinurie caractéristiques de la maladie de Berger sont dues à un dysfonctionnement glomérulaire consécutif au dépôt mésangial d’immunoglobulines (Ig) A1 anormales. Hypercellularité glomérulaire et glomérulosclérose sont régulièrement retrouvées sur les biopsies des patients avec maladie de Berger. L’anomalie majeure des IgA1 circulants est une modification de la O-glycosylation...