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This study was conducted on a storage reservoir situated in a valley, in the lower course of the Sząbruk stream in north-eastern Poland, the Olsztyn Lakeland mesoregion. The catchment area of the Sząbruk stream consists of an agricultural and an afforested part. A storage reservoir is found in the lower part of the Sząbruk stream valley. The reservoir was built 25 years ago. It is enclosed by a dike...
Evidence indicates that ischemia/reperfusion (IR) results in endothelial dysfunction and neutrophil adhesion in the post-ischemic myocardium and that ischemic preconditioning (IPC), superoxide dismutase (SOD), and anti-endothelin-1 (ET-1) interventions prevent these effects. We tested the hypothesis that ET-1-induced superoxide (O2-) generation mediates endothelial injury and neutrophil accumulation...
Evidence indicates that nitric oxide (NO) suppresses myocardial oxygen consumption (MVO2) and regulates myocardial substrate oxidation, however data from in vivo and isolated heart preparations are conflicting. In addition, cardiac endothelin (ET-1) release has been shown to increase with inhibition of NO synthase (NOS), however the effects of ET-1 on myocardial energetics is not clear. We employed...
Cardiac ischemia/reperfusion leads to coronary endothelial dysfunction, mediated by superoxide anion (O2-), but not hydroxyl radical (.OH). Ischemic preconditioning and mitochondrial ATP-dependent potassium channel opener (diazoxide) protect endothelium in the mechanism involving attenuation of O2- burst at reperfusion. We hypothesize that the endothelial protection involves upregulation of myocardial...
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