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This work aims to investigate the renal effect of hydrogen sulfide (H2S), in the experimentally induced diabetic nephropathy, besides the role of activation of АТP‐sensitive potassium (KАTP) channel in that effect. Thirty‐two adult male albino rats randomly divided into four groups: Control, streptozotocin‐induced diabetic (diabetic nephropathy [DN]), DN+NaHS (the H2S inducer), and DN+NaHS+Glibenclamide...