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New Findings
What is the central question of this study?
What is the cellular basis of the protection conferred on the heart by overexpression of caveolin‐3 (Cav‐3 OE) against many of the features of heart failure normally observed in vivo?
What is the main finding and its importance?
Cav‐3 overexpression has little effect in normal ventricular myocytes but reduces cellular hypertrophy and preserves...
New Findings
What is the central question of this study?
Heart failure is associated with redistribution of L‐type Ca2+ current (ICa) away from the t‐tubule membrane to the surface membrane of cardiac ventricular myocytes. However, the underlying mechanism and its dependence on severity of pathology (hypertrophy versus failure) are unclear.
What is the main finding and its importance?
Increasing...
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