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Retinopathy of prematurity (ROP) is characterized by deficits in the scotopic pathway, although the cellular locus for these deficits is not clear. Here we examined neurochemical and cellular changes that develop during oxygen‐induced retinopathy, a model of ROP. In addition, we examined whether treatment with the angiotensin II type‐1 receptor inhibitor, valsartan, prevented these changes. Newborn...