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Experimental crescentic glomerulonephritis is driven by systemic cellular immune responses. A pathogenic role for T helper type 1 (Th1) and Th17 cells is well established. T‐bet, a key transcription factor required for Th1 lineage commitment, and retinoic acid‐related orphan receptor‐γt (Rorγt), a key Th17 transcription factor, are required for full expression of disease. Similarly, several Th1‐ and...