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BackgroundSevoflurane post‐conditioning (SePost) has been found to alleviate ischemic myocardial reperfusion injury through the activation of prosurvival kinases. Lowered myocardial oxygen demand from reduced cardiac work may also contribute to cardioprotection, and is much less well‐studied. Our aim was to examine the simultaneous effects of SePost on cardiac work (here, rate–pressure product, RPP)...