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The complement system is involved in several aspects of the pathophysiology of myocardial ischemia and infarction. Initially a role for complement in ischemic heart disease was inferred from the deposition of complement components within the myocardium of experimental models of myocardial infarction. Further animal models demonstrated that depletion or inhibition of complement prior to myocardial...
The complement system is involved in several aspects of the pathophysiology of myocardial ischemia and infarction. Initially a role for complement in ischemic heart disease was inferred from the deposition of complement components within the myocardium of experimental models of myocardial infarction. Further animal models demonstrated that depletion or inhibition of complement prior to myocardial...
Strong evidence links cardiomyocyte loss to the pathology of some forms of heart failure. Both necrotic and apoptotic modes of cell death have been invoked as the mechanism underlying progressive cardiomyocyte dropout. Nitric oxide (NO) has received particular attention as a candidate reactive oxygen intermediate that influences not only cardiac function, but also cell death elicited by both apoptotic...
Strong evidence links cardiomyocyte loss to the pathology of some forms of heart failure. Both necrotic and apoptotic modes of cell death have been invoked as the mechanism underlying progressive cardiomyocyte dropout. Nitric oxide (NO) has received particular attention as a candidate reactive oxygen intermediate that influences not only cardiac function, but also cell death elicited by both apoptotic...
Strong evidence links cardiomyocyte loss to the pathology of some forms of heart failure. Both necrotic and apoptotic modes of cell death have been invoked as the mechanism underlying progressive cardiomyocyte dropout. Nitric oxide (NO) has received particular attention as a candidate reactive oxygen intermediate that influences not only cardiac function, but also cell death elicited by both apoptotic...
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