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After infection with Plasmodium chabaudi, C57BL/10 mice produced significant amounts of serum IFN-γ, developed a low level of parasitemia and survived the infection. Production of IFN-γ was also obvious when spleen cells of the infected mice were stimulated with the parasite antigen contained in an erythrocyte lysate in vitro. Depletion of CD4 + T cells abrogated production of IFN-γ, leading...