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Eur J Clin Invest 2011; 41 (11): 1210–1219
AbstractBackground In models of acute ischaemia, erythropoietin (EPO) administration has been found to attenuate vascular injury largely through reduced apoptosis, suppressed inflammation and increased nitric oxide availability. We studied the association between circulating endogenous EPO and no‐reflow in patients with first ST‐elevation myocardial infarction...
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