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Lipopolysaccharide (LPS; a model of systemic bacterial infection) causes fever and activates glucagon-like peptide-1 (GLP-1) neurons in the caudal brainstem. The present study examined whether central GLP-1 receptor signaling plays a functional role in LPS-induced fever. Adult male Sprague-Dawley rats were injected i.p. with LPS (0 or 100 μg/kg), then were infused intracerebroventricularly with GLP-1...