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The tumor suppressor p14arf interacts, in response to oncogenic signals, with the p53 E3‐ubiquitin ligase HDM2, thereby resulting in p53 stabilization and activation. In addition, it also exerts tumor‐suppressive functions in p53‐independent contexts. The activities of p14arf are regulated by the nucleolar chaperone nucleophosmin (NPM1), which controls its levels and cellular localization. In acute...
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