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TGFβ plays a critical role in tendon formation and healing. While its downstream effector Smad3 has been implicated in the healing process, little is known about the role of Smad3 in normal tendon development or tenocyte gene expression. Using mice deficient in Smad3 (Smad3−/−), we show that Smad3 ablation disrupts tendon architecture and has a dramatic impact on normal gene and protein expression...
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