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Background
The IL‐33/ST2 immune axis plays crucial roles in infection and immunity. A dysregulated IL‐33/ST2 axis can induce autoimmune reaction and inflammatory responses. Guillain‐Barré syndrome (GBS) is an acute peripheral neuropathy, mostly caused by post‐infection autoimmunity. The role of IL‐33/ST2 axis is not known in GBS. This study aimed to explore the role of IL‐33/ST2 axis in GBS.
Methods...