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Ethanol consumption impairs learning and memory through disturbances of NMDA‐type glutamate receptor‐dependent synaptic plasticity (long‐term depression [LTD] and long‐term potentiation [LTP]) in the hippocampus. Recently, we demonstrated that two ethanol binge‐like episodes in young adult rats selectively blocked NMDA‐LTD in hippocampal slices, increased NMDA receptor sensitivity to a GluN2B subunit...
Ethanol (EtOH) induces cognitive impairment through modulation of synaptic plasticity notably in the hippocampus. The cellular mechanism(s) of these EtOH effects may range from synaptic signaling modulation to alterations of the epigenome. Previously, we reported that two binge‐like exposures to EtOH (3 g/kg, ip, 9 h apart) in adolescent rats abolished long‐term synaptic depression (LTD) in hippocampus...
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