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Random clusters of decoupled cells caused by cell death in diseased cardiac tissue can increase structural heterogeneity and give rise to abnormal patterns of propagation. The objective of this study was to use a 2-D 0.6 cm × 0.6 cm randomly generated model of a ventricular monolayer (LRd membrane model) (dx=dy=10 µm) to study the relationships between the underlying cardiac microstructure, the pattern...
Spatial dispersion of action potential duration has been linked to arrhythmogenesis in the diseased heart; however, the complex dynamic between repolarization dynamics and microstructural heterogeneity in critical regimes is largely unknown. The objective of this study was to use microstructural computer models of adult monolayers to study the effect of abrupt tissue expansion and gap junction uncoupling...
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