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In rat pancreatic β cells, arachidonic acid (AA) triggered intracellular Ca 2+ release. This effect could be mimicked by eicosatetraynoic acid, indicating that AA metabolism is not required. The AA-mediated Ca 2+ signal was not affected by inhibition of ryanodine receptors or emptying of ryanodine-sensitive store but was reduced by ∼70% following the disruption of acidic stores (treatment...
Arachidonic acid (AA) is generated in the pancreatic islets during glucose stimulation. We investigated whether AA activated extracellular Ca 2+ entry in rat pancreatic β cells via a pathway that was independent of the activation of voltage-gated Ca 2+ channels. The AA triggered [Ca 2+ ] i rise did not involve activation of GPR40 receptors or AA metabolism. When cells...
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