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The aim of this study was to determine if prior heat shock and consequential heat shock protein (HSP) accumulation would inhibit viral infection. Confluent cultures of Madin-Darby bovine kidney cells were subjected to 30 mins of hyperthermia at 42°C then allowed to recover at 37°C for six hrs. During the six hrs, HSP concentrations increased by 232% in heat-shocked cultures. Following the recovery...