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The study aimed to determine the genetic variability in pitch pine (Pinus rigida Mill.) growing in the Niepołomice Forest (southern Poland). In the late nineteenth and early twentieth century, Adolf Cieślar of the Department of Forestry Research in Mariabrunn near Vienna, Austria established the experimental crops of pitch pine. During the study, 227 trees that grew in seven subunits were considered;...
In this study the genetic structure of Istebna and Tarnawa plus trees was studied and polymorphism of twenty four provenances of spruce tested in a IPTNS−IUFRO 1964/68 site in Krynica representing selected regions of the species occurrence was analyzed. The genetic diversity was estimated with seven isoenzyme systems encoded in eleven loci. The highest genetic polymorphism was shown by plus trees...
The results of previous studies have shown high breeding values of Beskidian spruce. The aim of the study was to assess the genetic structure of seventeen Norway spruce provenances from the Beskidy Mts. tested in IPTNSIUFRO 1964/68 experiment in Krynica, which survived after massive wind damage on the plots. Polymorphism of five isozyme systems encoded in five loci was determined. The highest value...
The genetic variation of Norway spruce provenances from fourteen geographical regions were tested in the IPTNS−IUFRO 1964/68 experiment in Krynica. The genetic structure of seven isozyme systems coded by eleven loci was described. Parameters of genetic polymorphism i.e. the average number of alleles per locus and observed heterozygosity were 1.47 and 0.12, respectively. The spruces from Belarus were...
In connection with the decline of spruce stands on the Krynica plots of IPTNS-IUFRO 1964/68, observed in the last decade and attributed to the so-called spiral disease, an attempt was made to determine whether the current health condition of trees is influenced by genotype (provenance). As shown by preliminary observations, the spruces differed in the degree of survival depending on provenance. The...
AbstractExpression of major histocompatibility complex (MHC) classII genes is controlled at the transcriptional level by at least four trans-acting genes, CIITA, RFXANK, RFX5, and RFXAP. Defects in these regulatory genes result in the absence of MHC classII molecule expression and, thereby, cause a combined immunodeficiency. MHC classII deficiency is inherited as an autosomal recessive trait. Since...
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