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The hepatobiliary disposition of rhodamine 123 (RH‐123) and its glucuronidated (RH‐Glu) and deacylated (RH‐110) metabolites were studied in an isolated perfused rat liver (IPRL) model in the presence and absence of P‐glycoprotein (P‐gp) and Mrp2 inhibitors. A single dose (180 µg) of RH‐123 was added to a recirculating perfusate in the absence (Control) or presence of cyclosporine A (CyA) or dibromosulfophthalein...