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Objective: Oxidative stress is a candidate mechanism for ethanol neuropathology in fetal alcohol spectrum disorders. Our overall objective was to investigate the hypothesis that ethanol neurotoxicity involves production of reactive oxygen species (ROS) with negative downstream consequences for mitochondrial membrane potential and neuron survival. Methods: We use P4 cerebellar granule neurons (CGNs)...
The developing central nervous system (CNS) is highly susceptible to ethanol, with acute or chronic exposure producing an array of anomalies and cell loss. Certain periods of vulnerability have been defined for various CNS regions, and are often followed by periods of relative ethanol resistance. In the present study, neonatal rats were acutely exposed to ethanol during a time when peak cell death...
The developing central nervous system is extremely sensitive to ethanol, with well-defined temporal periods of vulnerability. Many brain regions are particularly susceptible to ethanol during the early neonatal period, corresponding to the human third trimester, which represents a dynamic period of growth and differentiation. For this study, neonatal rats were acutely exposed to ethanol or control...
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