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The influence of endogenous cell NO production and NO derived from exogenous sources on oxidant injury to cultured fetal rat lung alveolar epithelium and an animal model of pulmonary oxidant injury was examined. Confluent fetal rat alveolar epithelial cell monolayers were stimulated to produce NO after treatment with a combination of cytokines (IL-1β, TNF-α, IFN-γ), LPS...
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