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Das Ausmaß der mikrovaskulären Schädigung spielt eine zentrale Rolle in der Pathogenese der Sepsis. Es wird durch endotheliale Dysfunktionen wie Schwellung, Deformierung und Apoptose mit teilweisem Ablösen der Endothelzellen von der Basalmembran bestimmt, die dann als zirkulierende Endothelzellen und endotheliale Mikropartikel vermehrt im Blut von Sepsispatienten gefunden wurden. Die Reparatur dieses...
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