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The aim of this study was to investigate the effect of adriamycin (ADR) in signaling activation of NF-κB in ADR-sensitive and -resistant GLC 4 human small-cell lung carcinoma. ADR activated NF-κB only in ADR-sensitive GLC 4 cells in a time- and dose-dependant manner by stimulating IκBα degradation after 4h. Activation of NF-κB in response to tumor necrosis factor was intact in both...
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