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Objective: In sinoatrial (SA) node cells, nitric oxide (NO) exerts a dual effect on the hyperpolarization-activated current, I f , i.e. in basal conditions NO enhances I f whereas in the presence of β-adrenergic stimulation it decreases it. Recent studies have shown that I f is present in ventricular myocytes from hypertrophied or failing hearts where it may promote abnormal...
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