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Hepatocyte cell death is a universal feature of inflammatory liver diseases. The observation that mice deficient in the activation of nuclear factor-κB (NF-κB) are not viable because of excessive hepatocyte apoptosis induced by tumor necrosis factor (TNF) made it crystal-clear that NF-κB plays a central role in protecting hepatocytes against TNF-induced cell death. Also during TNF-mediated liver injury,...
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