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Free radical attack on the sugar-phosphate backbone generates oxidized apurinic/apyrimidinic (AP) residues in DNA. 2′-deoxyribonolactone (dL) is a C1′-oxidized AP site damage generated by UV and γ-irradiation, and certain anticancer drugs. If not repaired dL produces G→A transitions in Escherichia coli. In the base excision repair (BER) pathway, AP endonucleases are the major enzymes responsible for...
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