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The mutation of Gly12 to Val12 in the Aα chains of human fibrinogen Rouen is associated with a delayed proteolytic release of fibrinopeptide A (FpA or Aα 1 to 16 of fibrinogen) by thrombin, leading to a bleeding disorder. Analogs of FpA and FpA Rouen have been designed that include a Pro15 to replace Val15 in natural FpA and to mimic the frequent occurrences of a proline residue at equivalent positions...
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