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Alzheimer’s disease (AD) is the most common dementing illness and is pathologically characterized by deposition of the 40–42 amino acid peptide, amyloid-β (Aβ), as senile plaques. It is well documented that brain inflammatory mechanisms mediated by reactive glia are activated in response to Aβ plaques. A number of reports further suggest that T-cells are activated in AD patients, and that these cells...
Alzheimer’s disease (AD), the most common form of dementia, is a progressive, degenerative disorder of the central nervous system. The major hallmarks of AD include selective neuronal cell death and the presence of amyloid deposits and neurofibrillary tangles. Apolipoprotein E (ApoE) has also been shown to colocalize with these neuropathological lesions. Here is reviewed the role of ApoE in AD. The...
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