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The inflammatory reaction following ischemic brain injury involves bioactive mediators released mainly from leukocytes. The aim of this in vitro-study was to evaluate possible modulatory actions of heparin on nitric oxide synthesis induced by proinflammatory cytokines. Rat microvascular brain endothelial cells were isolated from adult rat brains and treated with interferon-γ (IFN-γ) and interleukin-1β...
In the brain large amounts of nitric oxide are produced in response to various pathological stimuli such as infectious agents, ischemia and trauma. Although it is known that endothelial cells can express the inducible isoform of nitric oxide synthase (iNOS) upon activation, the impact of different cytokines on iNOS expression in rat microvascular endothelial cells remains unclear. We now investigated...
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