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Background
Small cell neuroendocrine (NE) carcinomas of the prostate classically lose androgen receptor (AR) expression, may harbor loss of the RB1, TP53, and PTEN tumor suppressor genes, and are associated with a poor prognosis. However usual‐type adenocarcinomas may also contain areas of NE differentiation, and in this context the molecular features and biological significance are less certain...
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