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BACKGROUNDGli2, a transcription factor in the Hedgehog pathway, is overexpressed in castrate‐resistant prostate cancer (PCa). Previously we showed that Gli2 overexpression increased transcriptional activity of androgen receptor (AR) and conferred androgen growth‐independence to normally growth‐dependent PCa cells. Here we localized the regions of AR‐Gli2 protein interaction and determined the domains...
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