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Although CAMKK2 is overexpressed in several cancers, its role and relevant downstream signaling pathways in gastric cancer (GC) are poorly understood. Treatment of AGS GC cells with a CAMKK2 inhibitor, STO‐609, resulted in decreased cell proliferation, cell migration, invasion, colony‐forming ability, and G1/S‐phase arrest. Quantitative phosphoproteomics in AGS cells with the CAMKK2 inhibitor led...
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