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Background & Aims
Pregnant women may transmit their metabolic phenotypes to their offspring, enhancing the risk for nonalcoholic fatty liver disease (NAFLD); however, the molecular mechanisms remain unclear.
Methods
Prior to pregnancy female mice were fed either a maternal normal‐fat diet (NF‐group, “no effectors”), or a maternal high‐fat diet (HF‐group, “persistent effectors”), or were transitioned...
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