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Fragile histidine trail (FHIT) is a tumor suppressor in response to DNA damage which has been deleted in various tumors. However, the signaling mechanisms and interactions of FHIT with regard to apoptotic proteins including p53 and p38 in the DNA damage‐induced apoptosis are not well described. In the present study, we used etoposide‐induced DNA damage in MCF‐7 as a model to address these crosstalks...
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