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Mitochondrial permeabilisation after NO donor application did not activate caspase‐9. We have studied the X‐linked apoptosis inhibitor (XIAP) and Aven protein content in NO‐treated Jurkat cells. The level of both proteins increased in NO‐treated cells. Thus the increase in XIAP and Aven content could be the cause of the lack of caspase‐9 activity after mitochondrial permeabilisation in NO‐treated...
Calprotectin (CP), an abundant heterodimeric cytosolic protein of neutrophils, conveys a variety of functions such as tumor cell growth arrest and antimicrobial activity. We investigated CP activity and its possible apoptosis‐inducing mechanism of action against an antiandrogen therapy‐resistance prostate cancer cell line LNCaP. Cell viability and Annexin V FITC assays were performed in order to investigate...
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