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Dyshomeostasis of trace elements have been implicated in the progression of Alzheimer's disease (AD), which is characterized by amyloid‐β (Aβ) plaques. Trace elements are particularly associated with the Aβ plaques. Metal‐protein attenuating compounds have been developed to inhibit metals from binding to Aβ proteins, which result in Aβ termination, in the hope of improving cognitive functioning. However,...
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