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Summary The signaling pathways leading to extracellular signal-regulated kinase (ERK) and p38 mitogen-activated protein kinase (MAPK) activation by N-formyl-Met-Leu-Phe (fMLP) or platelet activating factor (PAF) in human neutrophils were examined. Previously, we found that changes of intracellular Ca2+ ([Ca $$^{2+}]_{\rm i}$$ ) stimulated by PAF and fMLP were due to Ca2+ influx and internal Ca2+...
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